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An off-lattice model for the extracellular matrix of tendon and muscle is presented. The model includes two dimensions perpendicular to the tendon axis. The arrangement is based on a continuum model of aponeurosis without collagen fibrils. The model is extended by collagen fibril bundles. Each bundle is defined by its central axis in the tendon, thickness and the corresponding number of collagen molecules. A non-linear interaction is used to study the mechanical behavior. The simulations include the ultrasonic velocity in the fibrils and the ultrasonic attenuation through the collagen fibril bundles. The simulation for the ultrasonic velocity is in agreement with the experimental results and the ultrasonic attenuation can be explained by the asymmetry of the collagen fibril bundles.Salicylic and nitroglycerin enhanced the response to phenylpropanolamine.
The purpose of this study was to determine the mechanism of the interaction of glyceryl trinitrate (GTN), salicylic acid (SA), and nitroglycerin (NTG) with phenylpropanolamine (PPA). This was accomplished by measuring the adrenaline plasma concentrations that were induced by the administration of these drugs alone and in combination with PPA. Blood was obtained from subjects receiving GTN, SA, or NTG alone and in combination with PPA. PPA was given intravenously in a dose of 40 mg. Blood was drawn at baseline, 1, 2, 3, and 4 h following the PPA infusion. The administration of GTN alone resulted in an increase in heart rate, blood pressure, and adrenaline concentrations. These values were greater after the addition of SA to GTN, indicating enhanced cardiovascular effects (ECV) of GTN by SA. Neither NTG nor
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